Texas Mechanobiology Symposium


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October 25, 2015 at the National Center for Therapeutics Manufacturing at Texas A&M

About Our Program

The goal of the Texas Mechanobiology Symposium is to enhance networking between researchers and increase student exposure to cutting-edge research on the role of mechanics in molecular, cell and tissue biology. This inaugural one-day symposium will begin with the keynote lecture from Michael Sacks, followed by invited talks by experts in mechanobiology at all length scales. Oral presentations will not only showcase ongoing research in the region but also expose local students and faculty to the work from invited national and international leaders in the field. Finally, abstracts are being solicited for student poster sessions that will provide a venue for student research presentations and networking. Posters will be judged to identify and recognize the top student and postdoctoral presentations.

Sacks, Michael

Michael S. Sacks, Ph.D.


Professor Michael Sacks is the W. A. “Tex” Moncrief, Jr. Simulation-Based Engineering Science Chair and a world authority on cardiovascular biomechanics. His research focuses on the quantification and modeling of the structure-mechanical properties of native and engineered cardiovascular soft tissues. He is a leading international authority on the mechanical behavior and function of the native and replacement heart valves. He is also active in the biomechanics of engineered tissues, and in understanding the in-vitro and in-vivo remodeling processes from a functional biomechanical perspective. Sacks is director of the ICES Center for Cardiovascular Simulation and professor of biomedical engineering.

His research includes multi-scale studies of cell/tissue/organ mechanical interactions in heart valves and is particularly focused on determining the local stress environment for heart valve interstitial cells. Recent research has included developing novel multi-scale models of the mitral valve and bioprosthetic heart valves, as well as constitutive models of ventricular myocardium that allow for the separation of the individual contributions of the myocyte and connective tissue networks.